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Results Moderate and high overexpression of SLC7A11 exert opposite effects on H 2 O 2 -induced cell death We sought to understand whether SLC7A11 overexpression promotes cell death under NADPH-depleting conditions other than glucose starvation

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Pulmonary fibrosis (PF) and chronic obstructive pulmonary disease (COPD) (Phase Ⅱ), further induce endoplasmic reticulum stress and DNA damage, leading to cellular mutations that increase the risk of cancer and promote lung cancer (Phase Ⅲ)