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Moreover, as yet, relatively little is known about possible modifications to cell homeostasis and the nature of cellular responses when TH is transfected into cells that normally do not express or produce only relatively low levels of this enzyme
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Elevated ROS can activate signaling pathways, promote lipid peroxidation in hepatocytes, and cause liver cell death, generating reactive lipid species like 4‑hydroxy-2-nonenal and malondialdehyde
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