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Overexpression of a constitutively active ERO1 mutant incapable forming this regulatory disulfide elicits ER stress, but to a relatively modest extent compared with reducing agents like DTT (Hansen et al., 2012
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APX1 (L-ascorbate peroxidase), GSH2 (glutathione synthetase), GSTF6 (glutathione S-transferase F6), GSTT1 (glutathione S-transferase THETA 1), PER1 (1-Cys peroxiredoxin PER1), AT1G65820 (glutathione S-transferase), GSTF12 (glutathione S-transferase phi 12), GSTF2 (glutathione S-transferase F2), GSTF8 (glutathione S-transferase F8), and GSTU19 (glutathione S-transferase U19) proteins were predicted as the main interaction partners of Arabidopsis cytosolic GPX2
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