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ROS generated after the formation of ceramide or after an increase in energy metabolism induced by several receptor-interacting protein kinases (RIPs), either in the mitochondrial ETC and/or by NOXs, have been reported to enhance necroptosis 123,124,125
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However, it was also suggested that the association DOX–GSH significantly decreased the antineoplastic effectiveness of DOX in a dose-dependent manner, thus proving a disadvantageous effect of GSH use [32]
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