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Targeted inactivation of the insulin receptor gene in mouse 3T3-L1 fibroblasts via homologous recombination

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the Annals of the New York Academy of Sciences review of thymosin beta 4 covering both the preclinical dermal healing models and the two phase 2 clinical trials in stasis and pressure ulcers reported that thymosin beta 4 accelerated dermal healing across normal, steroid treated, diabetic and aged animal models, and that in two phase 2 trials of stasis and pressure ulcers it shortened healing by almost a month among the patients who did heal