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All 30 isolates were resistant to meropenem (MIC, 32–128 mg/L), imipenem (MIC, ≥16 mg/L), cefepime (MIC, ≥32 mg/L), ceftazidime (MIC, 16 to ≥64 mg/L), cefoperazone-sulbactam (MIC, ≥64 mg/L), piperacillin-tazobactam (MIC, ≥128 mg/L), and levofloxacin (MIC, ≥8 mg/L)

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Another reason for the inability of DA-Raf to inactivate Akt is attributed to that PI3K can be activated by Ras-independent mechanisms, such as trimeric G-protein Gβγ subunit-mediated activation (Engelman et al, 2006)