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Conditional removal of the inwardly rectifying potassium channel Kir4.1 from astrocytes leads to decreased potassium uptake, resulting in reduced synaptic responses to repetitive stimulation and post-tetanic potentiation (Sibille, Pannasch and Rouach, 2014)
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“Phasic” axons that fatigue in release of glutamate neurotransmitter have lower glutamate levels than “tonic” glutamate axons with greater glutamate levels [35]
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