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MOTS-c modulates the FOXO transcription factors and regulates cellular senescence, the state in which aged or damaged cells stop dividing but refuse to die, instead secreting a cocktail of inflammatory signals known as the senescence-associated secretory phenotype (SASP)

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Conclusion Across topical and tissue models, GHK-Cu is studied for potential roles in matrix support, wound-area remodeling signals, oxidative balance, and inflammation moderation